Publication	Data		Fish	Anatomy
Osborn et al., 2020	Fig. 2		noton1/n1	adaxial cell myod1 expression spatial pattern, abnormal post-vent region slow muscle cell decreased amount, abnormal
Mano et al., 2019	Fig. 3		WT + MO3-noto	epiphysis bsx expression decreased amount, abnormal epiphysis exorh expression absent, abnormal
Schredelseker et al., 2018	Fig. 5		noton1/n1 (AB/TL)	pineal complex bsx expression decreased amount, abnormal pineal complex otx5 expression decreased amount, abnormal
Khuansuwan et al., 2016	Fig. 1		noton1/+ ,   noton1/n1	epiphysis nr2e3 expression decreased amount, abnormal epiphysis tbx2b expression decreased amount, abnormal
	Fig. 3		noton1/n1; sqet11Et ,   noton1/+; sqet11Et ,   noton1/+ ,   noton1/n1	parapineal organ has extra parts of type parapineal organ neuron, abnormal parapineal organ neuron fate specification increased occurrence, abnormal pineal gland development process quality, abnormal
	Fig. 4		noton1/n1; sqet11Et	parapineal organ has extra parts of type parapineal organ neuron, abnormal parapineal organ neuron fate specification increased occurrence, abnormal
	Fig. 7		noton1/n1; sqet11Et	parapineal organ has extra parts of type parapineal organ neuron, abnormal parapineal organ neuron fate specification increased occurrence, abnormal
	Fig. 8		noton1/n1; sqet11Et	habenula bilateral symmetry, abnormal habenula development process quality, abnormal habenula left side kctd12.1 expression decreased amount, abnormal habenula right side kctd12.1 expression increased amount, abnormal parapineal organ has extra parts of type parapineal organ neuron, abnormal
Helker et al., 2015	Fig. 3		nototk241/tk241; y1Tg	angioblast cell migration from lateral mesoderm to midline arrested, abnormal angioblast cell migration from lateral mesoderm to midline process quality, abnormal
Hochgreb-Hägele et al., 2013	Fig. 6		pd24Tg + MO3-noto	determination of digestive tract left/right asymmetry decreased process quality, abnormal lateral mesoderm morphogenesis decreased process quality, abnormal lateral plate mesoderm bilateral symmetry, abnormal lateral plate mesoderm mislocalised, abnormal
Wong et al., 2012	Fig. 9		noton1/n1	endocardium morphogenesis disrupted, abnormal
Dal-Pra et al., 2011	Fig. 8		WT + MO2-noto	axial chorda mesoderm decreased width, abnormal floor plate disorganized, abnormal hypochord disorganized, abnormal notochord disorganized, abnormal paraxial mesoderm increased area, abnormal
	Fig. S5		WT + MO2-noto	axial chorda mesoderm decreased width, abnormal floor plate disorganized, abnormal hypochord aplastic, abnormal notochord aplastic, abnormal paraxial mesoderm increased area, abnormal paraxial mesoderm left side fused with paraxial mesoderm right side, abnormal somite fused with somite, abnormal
Zhang et al., 2010	Fig. 5		noton1/n1	melanocyte distended, abnormal
Chung et al., 2008	Fig. 3		noton1/n1; zf5Tg	pancreas development disrupted, abnormal pancreas endocrine cell decreased amount, abnormal
Snelson et al., 2008	Fig. 3		noton1/n1; zf104Tg	epiphysis decreased size, abnormal pineal gland development disrupted, abnormal
Hadzhiev et al., 2007	Fig. 6		noton1/n1; sb15Tg	caudal fin physical object quality, abnormal
Yin et al., 2007	Fig. 6		noton1/n1	adaxial cell mislocalised, abnormal
Phenotype Annotation (1994-2006)	Fig. for (la620)		notola620 (AB)	notochord quality, abnormal post-vent region quality, abnormal
	Fig. for (m614)		notom614/m614 (AB)	floor plate quality, abnormal nervous system quality, abnormal notochord quality, abnormal somite quality, abnormal whole organism lethal (sensu genetics), abnormal
	Fig. for (n1)		noton1/n1 (AB)	notochord quality, abnormal somite quality, abnormal whole organism lethal (sensu genetics), abnormal
	Fig. for (tk241)		nototk241/tk241	floor plate quality, abnormal nervous system quality, abnormal notochord quality, abnormal somite quality, abnormal whole organism lethal (sensu genetics), abnormal
	Fig. for (tm229)		nototm229/tm229	floor plate quality, abnormal nervous system quality, abnormal notochord quality, abnormal somite quality, abnormal whole organism lethal (sensu genetics), abnormal
Gamse et al., 2002	Fig. 3		noton1/n1	pineal complex decreased size, abnormal
Biemar et al., 2001	Fig. 6		nototk241/tk241	endocrine pancreas development decreased process quality, abnormal pancreatic bud has fewer parts of type pancreatic B cell, abnormal
Coutelle et al., 2001	Fig. 4		noton1/n1	adaxial cell immature, abnormal adaxial cell mislocalised, abnormal striated muscle cell differentiation delayed, abnormal
Lewis et al., 2001	Table 1		noton1/n1 (AB)	notochord aplastic, abnormal primary motor neuron decreased amount, abnormal somite shape, abnormal whole organism viability, abnormal
Majumdar et al., 2000	Fig. 1		noton1/n1 (AB)	cell migration involved in kidney development disrupted, abnormal podocyte mislocalised laterally, abnormal pronephric glomerulus increased amount, abnormal pronephric glomerulus mislocalised laterally, abnormal
	Fig. 2		noton1/n1 (AB)	podocyte mislocalised laterally, abnormal sclerotome absent, abnormal
	Fig. 3		noton1/n1 (AB)	cell migration involved in kidney development disrupted, abnormal pronephric glomerulus increased amount, abnormal pronephric glomerulus mislocalised laterally, abnormal
	Fig. 4		noton1/n1 (AB)	glomerular filtration decreased process quality, abnormal whole organism edematous, abnormal
	Fig. 5		noton1/n1 (AB)	cell migration involved in kidney development disrupted, abnormal pronephric glomerular capillary attached to common cardinal vein, abnormal pronephric glomerular capillary glomerular endothelium fenestra closed, abnormal pronephric glomerular capillary membrane detached from pronephric glomerular basement membrane, abnormal pronephric glomerulus increased amount, abnormal pronephric glomerulus malformed, abnormal pronephric glomerulus mislocalised laterally, abnormal
Dheen et al., 1999	Fig. 6		noton1/n1	notochord absent, abnormal
Gothilf et al., 1999	Fig. 4		noton1/n1	pineal complex aanat2 expression absent, abnormal
Schilling et al., 1999	Fig. 5		noton1/n1 (AB)	determination of digestive tract left/right asymmetry decreased process quality, abnormal determination of heart left/right asymmetry decreased process quality, abnormal heart tube bilateral symmetry, abnormal intestinal bulb primordium bilateral symmetry, abnormal
Beattie et al., 1997	Fig. 1		noton1/n1	secondary motor neuron anatomical region discontinuous, abnormal secondary motor neuron axon morphology, abnormal
	Fig. 2		noton1/n1	floor plate split, abnormal secondary motor neuron anatomical region split, abnormal
	Fig. 3		noton1/n1	spinal cord has fewer parts of type primary motor neuron, abnormal whole organism lacks all parts of type notochord, abnormal
	Fig. 4		noton1/n1	spinal cord has fewer parts of type primary motor neuron, abnormal spinal cord posterior region lacks all parts of type primary motor neuron, abnormal
	Fig. 5		noton1/n1	axial mesoderm development disrupted, abnormal
	Fig. 6		noton1/n1	floor plate split, abnormal whole organism lacks all parts of type notochord, abnormal
Glasgow et al., 1997	Fig. 6		noton1/n1	hypophysis cell increased amount, abnormal hypophysis increased size, abnormal
Schier et al., 1997	Fig. 8		noton1/n1 (AB)	notochord aplastic/hypoplastic, abnormal somite malformed, abnormal
Kelsh et al., 1996	Fig. 5		nototk241/tk241 ,   nototm229/tm229	melanocyte displaced to neural tube ventral surface, abnormal
Melby et al., 1996	Fig. 7		noton1/n1 (AB)	notochord cell differentiation process quality, abnormal
Odenthal et al., 1996	Fig. 1		nototk241/tk241	floor plate decreased size, abnormal floor plate formation disrupted, abnormal notochord absent, abnormal notochord formation disrupted, abnormal somite fused with somite, abnormal
	Table 1		nototm229/tm229	notochord aplastic, abnormal post-vent region somite increased distance post-vent region somite, abnormal somite medial side fused with somite medial side, abnormal
Solnica-Krezel et al., 1996	Fig. T1		notom614/m614 (AB)	floor plate decreased size, abnormal notochord aplastic, abnormal
Stemple et al., 1996	Fig. T1		notom614/m614 (AB)	axial chorda mesoderm aplastic, abnormal floor plate decreased size, abnormal
van Eeden et al., 1996	Table 1		nototk241/tk241 ,   nototm229/tm229	floor plate morphology, abnormal horizontal myoseptum hypoplastic, abnormal notochord morphology, abnormal whole organism movement behavioral quality, abnormal whole organism viability, abnormal
Halpern et al., 1995	Fig. 1		noton1/n1 (AB)	floor plate spinal cord region patchy, abnormal muscle displaced to axis, abnormal notochord absent, abnormal notochord cell differentiation disrupted, abnormal notochord development disrupted, abnormal somite development process quality, abnormal somite fused with somite, abnormal
	Fig. 3		noton1/n1 (AB)	axial chorda mesoderm cell fate commitment disrupted, abnormal notochord cell differentiation disrupted, abnormal somite development process quality, abnormal
	Fig. 6		noton1/n1 (AB)	axial mesoderm structural organization disrupted, abnormal