Publication	Data		Fish	Anatomy
Gong et al., 2017	Fig. 2		ml2Tg + CRISPR1-insm1a	CaP motoneuron axon decreased length, abnormal CaP motoneuron axon increased branchiness, abnormal CaP motoneuron decreased amount, abnormal CaP motoneuron distance CaP motoneuron, abnormal MiP motor neuron decreased amount, abnormal
	Fig. 3		ml2Tg + MO5-insm1a ,   ml2Tg + CRISPR1-insm1a	neuron differentiation decreased process quality, abnormal neuron undifferentiated, abnormal
	Fig. 4		ml2Tg + CRISPR1-insm1a	swimming behavior decreased process quality, abnormal swimming decreased occurrence, abnormal swimming decreased rate, abnormal
	Fig. 5		AB + MO5-insm1a	CaP motoneuron axon decreased length, abnormal CaP motoneuron axon increased branchiness, abnormal whole organism nkx6.1 expression decreased amount, abnormal whole organism olig2 expression decreased amount, abnormal
	Fig. S2		ml2Tg + CRISPR1-insm1a	whole organism decreased life span, abnormal
	Fig. S3		ml2Tg + CRISPR1-insm1a	CaP motoneuron decreased amount, abnormal MiP motor neuron decreased amount, abnormal
	Fig. S4		ml2Tg + MO5-insm1a	CaP motoneuron axon decreased length, abnormal CaP motoneuron axon increased branchiness, abnormal CaP motoneuron decreased amount, abnormal CaP motoneuron distance CaP motoneuron, abnormal MiP motor neuron decreased amount, abnormal
	Fig. S5		AB + MO5-insm1a	whole organism ascl1a expression decreased amount, abnormal whole organism ascl1a expression increased amount, abnormal whole organism ascl1b expression decreased amount, abnormal whole organism isl2a expression decreased amount, abnormal whole organism isl2a expression increased amount, abnormal whole organism mnx2a expression decreased amount, abnormal whole organism mnx2a expression increased amount, abnormal whole organism mnx2b expression decreased amount, abnormal whole organism shha expression increased amount, abnormal
	text only		ml2Tg + CRISPR1-insm1a	thigmotaxis decreased process quality, abnormal
He et al., 2017	Fig. 2		zf106Tg + MO4-tp53 + MO5-insm1a	neuromast spatial pattern, abnormal posterior lateral line neuromast hair cell morphogenesis disrupted, abnormal posterior lateral line primordium neuromast decreased amount, abnormal
	Fig. 3		s356tTg + MO4-tp53 + MO5-insm1a	neuromast hair cell decreased amount, abnormal neuromast hair cell development disrupted, abnormal
	Fig. 4		s356tTg + MO4-tp53 + MO5-insm1a	posterior lateral line primordium cell population proliferation disrupted, abnormal posterior lateral line primordium decreased size, abnormal posterior lateral line primordium morphology, abnormal
	Fig. 5		WT + MO4-tp53 + MO5-insm1a	myoseptum cxcl12a expression decreased amount, abnormal posterior lateral line primordium ackr3b expression decreased amount, abnormal posterior lateral line primordium cxcr4b expression increased amount, abnormal
	Fig. 6		WT + MO4-tp53 + MO5-insm1a	posterior lateral line primordium axin2 expression increased distribution, abnormal posterior lateral line primordium etv4 expression decreased amount, abnormal posterior lateral line primordium lef1 expression increased distribution, abnormal regulation of canonical Wnt signaling pathway disrupted, abnormal
	Fig. S2		zf106Tg + MO4-tp53 + MO5-insm1a	posterior lateral line primordium neuromast decreased amount, abnormal
Forbes-Osborne et al., 2013	Fig. 2		WT + MO3-insm1a	eye decreased size, abnormal
	Fig. 3		WT + MO3-insm1a	retinal cone cell decreased amount, abnormal retinal cone cell differentiation disrupted, abnormal retinal rod cell decreased amount, abnormal retinal rod cell differentiation disrupted, abnormal
	Fig. 4		WT + MO3-insm1a ,   q16aTg; q16bTg + MO3-insm1a	retinal bipolar neuron decreased amount, abnormal retinal ganglion cell decreased amount, abnormal
	Fig. 5		WT + MO3-insm1a	horizontal cell immature, abnormal neuron maturation delayed, abnormal retinal bipolar neuron decreased amount, abnormal retinal bipolar neuron immature, abnormal retinal outer plexiform layer structure, abnormal
	Fig. 6		WT + MO3-insm1a	cell cycle process temporally extended, abnormal