Publication	Data		Fish	Anatomy
Arribat et al., 2020	Fig 2		spastula1/ula1	aerobic respiration decreased magnitude, abnormal hatching delayed, abnormal whole organism decreased length, abnormal whole organism decreased weight, abnormal
	Fig 3		spastula1/ula1	fast muscle cell endoplasmic reticulum aggregated, abnormal slow muscle cell endoplasmic reticulum flattened, abnormal swim bladder anatomical region increased accumulation fat cell, abnormal
	Fig 7		spastula1/ula1	brain atl1 expression increased amount, abnormal brain reep1 expression increased amount, abnormal skeletal muscle atl1 expression increased amount, abnormal skeletal muscle bscl2 expression increased amount, abnormal skeletal muscle reep1 expression increased amount, abnormal skeletal muscle spartb expression increased amount, abnormal
	Fig 8		spastula1/ula1	brain N-octadecanoylsphingosine decreased amount, abnormal brain phosphatidylethanolamine 34:0 zwitterion increased amount, abnormal brain phosphatidylethanolamine 36:1 zwitterion increased amount, abnormal brain phosphatidylethanolamine 36:2 zwitterion increased amount, abnormal brain phosphatidylethanolamine 40:3 zwitterion increased amount, abnormal skeletal muscle cholesteryl ester decreased amount, abnormal skeletal muscle hexadecanoic acid decreased amount, abnormal skeletal muscle hexadecatrienoic acid decreased amount, abnormal skeletal muscle N-docosanoylsphingosine decreased amount, abnormal
Jardin et al., 2018	Fig. 2		WT + MO4-spast ,   WT + MO3-spast	caudal fin curved, abnormal eye decreased size, abnormal thigmotaxis process quality, abnormal yolk agenesis, abnormal
	Fig. 3		ml2Tg + MO4-spast ,   ml2Tg + MO3-spast	secondary motor neuron axon guidance process quality, abnormal secondary motor neuron cell body mislocalised, abnormal
	Fig. 5		spastzf2203/+; ml2Tg (TU) ,   spastzf2203/+ (TU) ,   spastzf2203/zf2203 (TU)	secondary motor neuron axon guidance process quality, abnormal secondary motor neuron cell body mislocalised, abnormal thigmotaxis process quality, abnormal whole organism decreased size, abnormal
	Fig. 6		WT + MO3-spast	secondary motor neuron axon guidance process quality, abnormal
	Fig. 7		ml2Tg + MO4-spast	secondary motor neuron axon guidance process quality, abnormal secondary motor neuron cell body mislocalised, abnormal
Julien et al., 2016	Fig. 9		WT + MO3-spast	extension morphology, abnormal head hydrocephalic, abnormal motor neuron axon microtubule bundle decreased diameter, abnormal reactive oxygen species metabolic process process quality, abnormal spinal cord microtubule cytoskeleton disorganized, abnormal spinal cord microtubule cytoskeleton organization disrupted, abnormal whole organism arched, abnormal
Allison et al., 2013	Fig. 8		AB + MO3-spast	axon extension disrupted, abnormal spinal cord motor neuron branchiness, abnormal spinal cord motor neuron truncated, abnormal whole organism decreased mobility, abnormal whole organism increased curvature, abnormal
Zhang et al., 2012	Fig. 6		AB + MO2-spast	caudal fin morphology, abnormal extension morphology, abnormal head morphology, abnormal primary motor neuron axon morphology, abnormal primary motor neuron axonogenesis process quality, abnormal
Butler et al., 2010	Fig. 2		WT + MO2-spast + MO4-tp53	axon extension decreased occurrence, abnormal motor neuron axon decreased length, abnormal
	Fig. 3		WT + MO1-spast ,   WT + MO2-spast	cytoplasmic microtubule organization process quality, abnormal microtubule severing decreased occurrence, abnormal
	Fig. 4		WT + MO1-spast	axon extension decreased occurrence, abnormal motor neuron axon decreased length, abnormal
	Fig. 6		WT + MO1-spast	axon extension decreased occurrence, abnormal filopodium assembly process quality, abnormal
	Fig. 7		WT + MO1-spast	axon extension decreased occurrence, abnormal motor neuron axon decreased length, abnormal