FIGURE
            Fig. S1
- ID
- ZDB-FIG-071228-6
- Publication
- Grzmil et al., 2007 - The INT6 Cancer Gene and MEK Signaling Pathways Converge during Zebrafish Development
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                        Fig. S1
                    
                    
                
                
            
        
        
    
        
            
            
| Cell cycle analysis of int6 morphants. Whole-mount immunohistochemistry with the late G2/M phase marker, phospho-histone H3 shows only slightly reduced numbers of cells in late G2/M phase in the int6 morphant compared to the control. Similarly, DNA content as measured by flow cytometry reveals only a slight reduction of cells in G2/M phase in the int6 morphant. Thus, we find that loss of Int6 in normal vertebrate cells (as well as in additional human cancer cell lines, M.G. & C.J.N. unpublished data) does not appear result in an accumulation of cells in G2/M progression. | 
                
                    
                        Expression Data
                    
                    
                
                
            
        
        
    
        
            
            
            
            
    
    
                
                    
                        Expression Detail
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Antibody Labeling
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Phenotype Data
                    
                    
                
                
            
        
        
    
        
            
            
            
            
    
    
                
                    
                        Phenotype Detail
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Acknowledgments
                    
                    
                
                
            
        
        
    
        
            
            
                
                    
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      Full text @ PLoS One
                
                
            
        
        
    
    
     
        