FIGURE
            Fig. 4
- ID
- ZDB-FIG-071008-10
- Publication
- Grzmil et al., 2007 - The INT6 Cancer Gene and MEK Signaling Pathways Converge during Zebrafish Development
- Other Figures
- All Figure Page
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                        Fig. 4
                    
                    
                
                
            
        
        
    
        
            
            
| Int6 and Mek signaling interact in vivo. (A–C). Only embryos treated with the 1.0 μM CI-1040, and not 0.25 μM, show a loss of posterior structures, in contrast to (D) int6 morphants (int6 MO 0.25 ng). (E, F) In combination with 0.25 μM of CI-1040, 0.25 μg of int6 MO causes a dramatic alteration of the anterior-posterior axis. | 
                
                    
                        Expression Data
                    
                    
                
                
            
        
        
    
        
            
            
            
            
    
    
                
                    
                        Expression Detail
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Antibody Labeling
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Phenotype Data
                    
                    
                
                
            
        
        
    
        
            
            
            
            
    
    
                
                    
                        Phenotype Detail
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Acknowledgments
                    
                    
                
                
            
        
        
    
        
            
            
                
                    
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      Full text @ PLoS One
                
                
            
        
        
    
    
     
        