Publication	Data		Fish	Anatomy
Allen et al., 2020	Fig. 1		WT + MO2-acvr1l + MO4-acvr1l	whole organism anterior-posterior axis increased length, abnormal whole organism dead, abnormal whole organism dorsalized, abnormal whole organism viability, abnormal
	Fig. 2		WT + MO2-acvr1l + MO4-acvr1l	blastoderm ventral region Ab13-smad labeling decreased distribution, abnormal
Ferrero et al., 2018	Fig. 2		acvr1ltm110b/tm110b	brain microglial cell apoeb expression absent, abnormal brain microglial cell absent, abnormal
Huang et al., 2018	Fig. 4		WT + MO3-acvr1l	anterior lateral plate mesoderm spi1b expression decreased distribution, abnormal
Mucha et al., 2018	Fig. 1		acvr1ltm110b/tm110b	whole organism dorsalized, abnormal
Umasankar et al., 2012	Fig. 5		WT + MO2-acvr1l	whole organism wholly dorsalized, abnormal
Joziasse et al., 2011	Fig. 3		acvr1ltm110b/tm110b	whole organism wholly dorsalized, abnormal
Liu et al., 2010	Fig. 2		acvr1ltm110b/tm110b	epicardium aplastic, abnormal
Little et al., 2009	Fig. 2		WT + MO2-acvr1l + MO4-acvr1l	whole organism wholly dorsalized, abnormal
Marques et al., 2009	Fig. 1		acvr1lsk42/sk42 ,   acvr1lsk42/sk42; f2Tg	atrium cardiac muscle cell decreased amount, abnormal atrium deformed, abnormal cardiac ventricle deformed, abnormal
	Fig. 3		acvr1lsk42/sk42	anterior lateral plate mesoderm decreased width, abnormal
Shen et al., 2009	Fig. 3		acvr1ltm110b/tm110b	dorsal/ventral pattern formation disrupted, abnormal ventral fin fold aplastic, abnormal whole organism wholly dorsalized, abnormal
Smith et al., 2008	Fig. 5		acvr1ltm110b/tm110b	embryonic heart tube development disrupted, abnormal heart rudiment rotated, abnormal
Chocron et al., 2007	Fig. 1		acvr1ltm110b/tm110b (TU)	atrium increased width, abnormal atrium position, abnormal diencephalon bilateral, abnormal heart looping arrested, abnormal
	text only		acvr1ltm110b/tm110b (TU)	blood circulation arrested, abnormal determination of ventral identity process quality, abnormal heart edematous, abnormal
Song et al., 2007	Fig. 2		AB + MO3-acvr1l	liver aplastic, abnormal pancreas aplastic, abnormal pancreatic B cell decreased amount, abnormal
Phenotype Annotation (1994-2006)	Fig. for (gl2)		acvr1lgl2/gl2	fin decreased size, abnormal hemopoiesis quality, abnormal
	Fig. for (m100)		acvr1lm100/m100 (AB)	surface structure quality, abnormal
	Fig. for (tm110b)		acvr1ltm110b/tm110b (TU)	fin decreased size, abnormal pectoral fin quality, abnormal surface structure quality, abnormal whole organism lethal (sensu genetics), abnormal
Bauer et al., 2001	Fig. 2		WT + MO2-acvr1l	post-vent region truncated, abnormal whole organism wholly dorsalized, abnormal
	Fig. 3		acvr1ltm110b/tm110b (TU)	caudal fin ventral side aplastic, abnormal pericardial cavity increased size, abnormal
Mullins et al., 1996	Fig. 2		acvr1ltm110b/tm110b (TU)	anal fin absent, abnormal blood circulation disrupted, abnormal caudal vein absent, abnormal convergent extension involved in axis elongation process quality, abnormal tail bud increased size, abnormal ventricular system aplastic, abnormal
	Fig. 3		acvr1ltm110b/tm110b (TU)	post-vent region somite increased width, abnormal
	Fig. 4		acvr1ltm110b/tm110b (TU)	erythroid lineage cell decreased amount, abnormal
Solnica-Krezel et al., 1996	Fig. 1		acvr1lm100/m100 (AB)	caudal fin lower lobe mislocalised, abnormal ventral fin fold morphology, abnormal
	Fig. 4		acvr1lm100/m100 (AB)	Kupffer's vesicle dislocated yolk, abnormal tail bud everted, abnormal
	Fig. 5		acvr1lm100/m100 (AB)	caudal vein plexus absent, abnormal ventral fin fold absent, abnormal
	Fig. T1		acvr1lm100/m100 (AB)	heart morphology, abnormal post-vent region decreased length, abnormal
van Eeden et al., 1996	Table 1		acvr1ltm110b/tm110b	caudal fin ventral region hypoplastic, abnormal whole organism wholly dorsalized, abnormal
	Table 2		acvr1ltm110b/tm110b	median fin fold hypoplastic, abnormal