FIGURE
            Figure 6
- ID
- ZDB-FIG-230529-89
- Publication
- Shi et al., 2023 - Deletion of the foxO4 Gene Increases Hypoxia Tolerance in Zebrafish
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                        Figure 6
                    
                    
                
                
            
        
        
    
        
            
            
| 
 NAD(H) content and expression of mitochondrial respiratory function-regulated genes. NADH (A) and NAD+ (B) contents and NADH/NAD+ rate (C) in muscle tissue of foxO4−/− and WT adult zebrafish. Expression of genes associated with mitochondrial respiratory complexes I (D), III (E), IV (F) and V (G). Gene expression level was normalized to ef1a. ** p < 0.01, *** p < 0.001. | 
                
                    
                        Expression Data
                    
                    
                
                
            
        
        
    
        
            
            
    | Genes: | |
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| Fish: | |
| Anatomical Term: | |
| Stage: | Adult | 
                
                    
                        Expression Detail
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Antibody Labeling
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Phenotype Data
                    
                    
                
                
            
        
        
    
        
            
            
  
    | Fish: | |
|---|---|
| Observed In: | |
| Stage: | Adult | 
                
                    
                        Phenotype Detail
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Acknowledgments
                    
                    
                
                
            
        
        
    
        
            
            
                
                    
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      Full text @ Int. J. Mol. Sci.
                
                
            
        
        
    
    
     
        