FIGURE
            Fig. 2
- ID
- ZDB-FIG-110112-16
- Publication
- Lin et al., 2010 - Loss of Cofilin 1 Disturbs Actin Dynamics, Adhesion between Enveloping and Deep Cell Layers and Cell Movements during Gastrulation in Zebrafish
- Other Figures
- All Figure Page
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                        Fig. 2
                    
                    
                
                
            
        
        
    
        
            
            
| Knockdown of cfl1 causes epiboly defects. Embryos were injected with or without cfl1 MO and photographed at 10 h post-fertilization (hpf). The cfl1 MO caused epiboly defects of different severities. (A) A sham-injected embryo reached 100% epiboly. (B) A cfl1 morphant with a malformed tail bud. (C) A cfl1 morphant that reached 90% epiboly. (D) A cfl1 morphant that reached 50% epiboly. Scale bar, 200 μm. | 
                
                    
                        Expression Data
                    
                    
                
                
            
        
        
    
        
            
            
            
            
    
    
                
                    
                        Expression Detail
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Antibody Labeling
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Phenotype Data
                    
                    
                
                
            
        
        
    
        
            
            
  
    | Fish: | |
|---|---|
| Knockdown Reagent: | |
| Observed In: | |
| Stage: | Bud | 
                
                    
                        Phenotype Detail
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Acknowledgments
                    
                    
                
                
            
        
        
    
        
            
            
                
                    
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      Full text @ PLoS One
                
                
            
        
        
    
    
     
        