Morpholino

MO2-nme2b.1

ID
ZDB-MRPHLNO-091119-1
Name
MO2-nme2b.1
Previous Names
  • MO2-nme2
Target
Sequence
5' - GGTGCGCTCGGTCTTAGCAGACATG - 3'
Disclaimer
Although ZFIN verifies reagent sequence data, we recommend that you conduct independent sequence analysis before ordering any reagent.
Note
This is a translation blocking morpholino.
Genome Resources
None
Target Location
Genomic Features
No data available
Expression
Gene expression in Wild Types + MO2-nme2b.1
Phenotype
Phenotype resulting from MO2-nme2b.1
Phenotype Fish Figures
angiogenesis disrupted, abnormal y1Tg + MO2-nme2b.1 Fig. S1 from Feng et al., 2014
brain vasculature malformed, abnormal y1Tg + MO2-nme2b.1 Fig. S1 from Feng et al., 2014
cAMP biosynthetic process decreased rate, abnormal f1Tg + MO2-nme2b.1 Fig. 5 with image from Hippe et al., 2009
central artery malformed, abnormal y1Tg + MO2-nme2b.1 Fig. S1 from Feng et al., 2014
dorsal longitudinal anastomotic vessel malformed, abnormal y1Tg + MO2-nme2b.1 Fig. 1 from Feng et al., 2014
eye decreased size, abnormal WT + MO2-nme2b.1 Fig. 2 with image from Hippe et al., 2009
GTP biosynthetic process decreased rate, abnormal WT + MO2-nme2b.1 Fig. 1 with image from Hippe et al., 2009
heart composition, abnormal f1Tg + MO2-nme2b.1 Fig. 5 with image from Hippe et al., 2009
heart decreased contractility, abnormal WT + MO2-nme2b.1 Fig. 2 with image from Hippe et al., 2009
heart decreased functionality, abnormal WT + MO2-nme2b.1 Fig. 2 with image from Hippe et al., 2009
heart contraction decreased rate, abnormal WT + MO2-nme2b.1 Fig. 2 with image from Hippe et al., 2009
heart contraction process quality, abnormal WT + MO2-nme2b.1 Fig. 1 from Hippe et al., 2011
intersegmental vessel malformed, abnormal y1Tg + MO2-nme2b.1 Fig. 1 from Feng et al., 2014
parachordal vessel aplastic, abnormal y1Tg + MO2-nme2b.1 Fig. 1 from Feng et al., 2014
pericardium edematous, abnormal WT + MO2-nme2b.1 Fig. 1 from Hippe et al., 2011
Fig. 2 with image from Hippe et al., 2009
subintestinal vein absent, abnormal y1Tg + MO2-nme2b.1 Fig. 1 from Feng et al., 2014
subintestinal vein malformed, abnormal y1Tg + MO2-nme2b.1 Fig. 1 from Feng et al., 2014
whole organism ab1-gnb labeling decreased amount, abnormal WT + MO2-nme2b.1 Fig. 1 from Hippe et al., 2011
Phenotype of all Fish created by or utilizing MO2-nme2b.1
Phenotype Fish Conditions Figures
whole organism ab1-gnb labeling decreased amount, abnormal WT + MO2-nme2b.1 control Fig. 1 from Hippe et al., 2011
heart decreased contractility, abnormal WT + MO2-nme2b.1 standard conditions Fig. 2 with image from Hippe et al., 2009
pericardium edematous, abnormal WT + MO2-nme2b.1 control Fig. 1 from Hippe et al., 2011
Fig. 2 with image from Hippe et al., 2009
heart contraction decreased rate, abnormal WT + MO2-nme2b.1 standard conditions Fig. 2 with image from Hippe et al., 2009
heart contraction process quality, abnormal WT + MO2-nme2b.1 control Fig. 1 from Hippe et al., 2011
heart decreased functionality, abnormal WT + MO2-nme2b.1 standard conditions Fig. 2 with image from Hippe et al., 2009
eye decreased size, abnormal WT + MO2-nme2b.1 standard conditions Fig. 2 with image from Hippe et al., 2009
GTP biosynthetic process decreased rate, abnormal WT + MO2-nme2b.1 standard conditions Fig. 1 with image from Hippe et al., 2009
heart composition, abnormal f1Tg + MO2-nme2b.1 standard conditions Fig. 5 with image from Hippe et al., 2009
cAMP biosynthetic process decreased rate, abnormal f1Tg + MO2-nme2b.1 standard conditions Fig. 5 with image from Hippe et al., 2009
subintestinal vein absent, abnormal y1Tg + MO2-nme2b.1 standard conditions Fig. 1 from Feng et al., 2014
subintestinal vein malformed, abnormal y1Tg + MO2-nme2b.1 standard conditions Fig. 1 from Feng et al., 2014
central artery malformed, abnormal y1Tg + MO2-nme2b.1 standard conditions Fig. S1 from Feng et al., 2014
intersegmental vessel malformed, abnormal y1Tg + MO2-nme2b.1 standard conditions Fig. 1 from Feng et al., 2014
parachordal vessel aplastic, abnormal y1Tg + MO2-nme2b.1 standard conditions Fig. 1 from Feng et al., 2014
brain vasculature malformed, abnormal y1Tg + MO2-nme2b.1 standard conditions Fig. S1 from Feng et al., 2014
dorsal longitudinal anastomotic vessel malformed, abnormal y1Tg + MO2-nme2b.1 standard conditions Fig. 1 from Feng et al., 2014
angiogenesis disrupted, abnormal y1Tg + MO2-nme2b.1 standard conditions Fig. S1 from Feng et al., 2014
cardiac ventricle heart contraction process quality, abnormal WT + MO1-nme3 + MO2-nme2b.1 standard conditions Fig. 6 from Abu-Taha et al., 2017
Citations