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Fig. S6
Cell non-autonomous Induction of Cyp26a1 overexpression is not responsible for a loss of RA signaling. (A–A3) Cyp26a1 deficient Tg(β-actin:GDBD-RLBD);(UAS:EGFP) donor cells transplanted into WT hosts have bright EGFP expression. (B–B3) Cyp26a1 deficient Tg(β-actin:GDBD-RLBD);(UAS:EGFP) donor cells transplanted into vp-rar mRNA injected hosts have dampened EGFP expression.
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Reprinted from Developmental Biology, 405(1), Rydeen, A., Voisin, N., D'Aniello, E., Ravisankar, P., Devignes, C.S., Waxman, J.S., Excessive feedback of Cyp26a1 promotes cell non-autonomous loss of retinoic acid signaling, 47-55, Copyright (2015) with permission from Elsevier. Full text @ Dev. Biol.