Publication	Data		Fish	Anatomy
Zheng et al., 2009	Fig. 1		WT + MO1-cald1b	blood vessel morphogenesis disrupted, abnormal mandibular arch skeleton morphology, abnormal skeletal system morphogenesis disrupted, abnormal trunk vasculature morphology, abnormal
	Fig. 2		WT + MO1-cald1b	blood circulation disrupted, abnormal blood vessel endothelial cell migration disrupted, abnormal blood vessel morphogenesis disrupted, abnormal caudal vein plexus decreased size, abnormal dorsal aorta morphology, abnormal dorsal longitudinal anastomotic vessel aplastic, abnormal intersegmental vessel decreased amount, abnormal intersegmental vessel morphology, abnormal morphogenesis of an epithelial sheet disrupted, abnormal nucleate erythrocyte aggregated, abnormal nucleate erythrocyte fluid flow rate, abnormal posterior cardinal vein morphology, abnormal subintestinal vein morphology, abnormal trunk vasculature morphology, abnormal
	Fig. 4		WT + MO1-cald1b	blood vessel morphogenesis disrupted, abnormal cranial vasculature dilated, abnormal cranial vasculature morphology, abnormal
	Fig. 5		WT + MO1-cald1b	aortic arch morphology, abnormal blood vessel morphogenesis disrupted, abnormal
	text only		WT + MO1-cald1b	blood circulation disrupted, abnormal nucleate erythrocyte decreased amount, abnormal trunk vasculature broken, abnormal trunk vasculature morphology, abnormal
Zheng et al., 2009	Fig. 1		WT + MO1-cald1b ,   WT + MO2-cald1b	atrioventricular valve aplastic, abnormal atrium elongated, abnormal atrium increased size, abnormal cardiac muscle cell decreased amount, abnormal cardiac muscle cell decreased size, abnormal cardiac muscle cell proliferation disrupted, abnormal cardiac ventricle elongated, abnormal heart looping disrupted, abnormal heart morphogenesis disrupted, abnormal heart morphology, abnormal pericardium edematous, abnormal
	text only		WT + MO1-cald1b ,   WT + MO2-cald1b	blood circulation disrupted, abnormal cardiac muscle cell proliferation disrupted, abnormal heart contraction decreased rate, abnormal heart contraction irregular rhythm, abnormal heart looping disrupted, abnormal heart morphogenesis disrupted, abnormal heart morphology, abnormal pericardium edematous, abnormal
Zheng et al., 2009	Fig. 1		WT + MO1-cald1b ,   WT + MO2-cald1b	angiogenesis disrupted, abnormal blood circulation decreased occurrence, abnormal subintestinal vein decreased functionality, abnormal subintestinal vein structure, abnormal
Zheng et al., 2009	Fig. 1		WT + MO1-cald1b ,   WT + MO2-cald1b	cardiac conduction system development disrupted, abnormal heart contractility, abnormal heart contraction decreased rate, abnormal heart contraction irregular rhythm, abnormal heart decreased functionality, abnormal