ZFIN ID: ZDB-PERS-051122-1
Danesin, Cathy
Email: cathy.danesin@univ-tlse3.fr
Affiliation: Soula Lab
Address: Centre de Biologie du Développement UMR5547 CNRS/Université Paul Sabatier 118 route de Narbonne - Bât. 4R3 b3 31062 Toulouse cedex 9, France
Country: France

1) Regulation of signalling factors during neural development

2) Oligodendrocyte development

3) Extracellular sulfatase Sulf1

Danesin, C., Darche-Gabinaud, R., Escalas, N., Bouguetoch, V., Cochard, P., Al Oustah, A., Ohayon, D., Glise, B., Soula, C. (2021) Sulf2a controls Shh-dependent neural fate specification in the developing spinal cord. Scientific Reports. 11:118
Danesin, C., Soula, C. (2017) Moving the Shh Source over Time: What Impact on Neural Cell Diversification in the Developing Spinal Cord?. Journal of developmental biology. 5(2):4
Al Oustah, A., Danesin, C., Khouri-Farah, N., Farreny, M.A., Escalas, N., Cochard, P., Glise, B., and Soula, C. (2014) Dynamics of Sonic hedgehog signaling in the ventral spinal cord are controlled by intrinsic changes in source cells requiring Sulfatase 1. Development (Cambridge, England). 141(6):1392-1403
Danesin, C., and Houart, C. (2012) A Fox stops the Wnt: implications for forebrain development and diseases. Current opinion in genetics & development. 22(4):323-330
Paridaen, J.T., Danesin, C., Elas, A.T., van de Water, S., Houart, C., and Zivkovic, D. (2009) Apc1 is required for maintenance of local brain organizers and dorsal midbrain survival. Developmental Biology. 331(2):101-112
Danesin, C., Peres, J.N., Johansson, M., Snowden, V., Cording, A., Papalopulu, N., and Houart, C. (2009) Integration of telencephalic Wnt and hedgehog signaling center activities by Foxg1. Developmental Cell. 16(4):576-587
Paridaen, J.T., Danesin, C., Elas, A.T., van de Water, S., Houart, C., and Zivkovic, D. (2009) Apc1-Mediated Antagonism of Wnt/beta-Catenin Signaling Is Required for Retino-Tectal Pathfinding in the Zebrafish. Zebrafish. 6(1):41-47

Touahri Y, Escalas N, Benazeraf B, Cochard P, Danesin C, Soula C.
Sulfatase 1 promotes the motor neuron-to-oligodendrocyte fate switch by activating Shh signaling in Olig2 progenitors of the embryonic ventral spinal cord. J Neurosci. 2012 Dec 12;32(50):18018-34