Morpholino

MO1-psenen

ID
ZDB-MRPHLNO-060331-1
Name
MO1-psenen
Previous Names
  • Pen-2 MO (1)
Target
Sequence
5' - CATAATGAAGTATTTGTGGTGGTGG - 3'
Disclaimer
Although ZFIN verifies reagent sequence data, we recommend that you conduct independent sequence analysis before ordering any reagent.
Note
None
Genome Resources
None
Target Location
Genomic Features
No data available
Expression
Gene expression in Wild Types + MO1-psenen
Phenotype
Phenotype resulting from MO1-psenen
Phenotype Fish Figures
brain lacks parts or has fewer parts of type ventricular system, abnormal WT + MO1-psenen Fig. 9 from Campbell et al., 2006
brain primary motor neuron decreased amount, abnormal WT + MO1-psenen + MO4-tp53 Fig. 9 from Campbell et al., 2006
brain primary motor neuron spatial pattern, abnormal WT + MO1-psenen + MO4-tp53 Fig. 9 from Campbell et al., 2006
caudal fin increased pigmentation, abnormal WT + MO1-psenen Fig. 3 from Ralser et al., 2017
head decreased pigmentation, abnormal WT + MO1-psenen Fig. 3 from Ralser et al., 2017
head decreased size, abnormal WT + MO1-psenen Fig. 3 from Campbell et al., 2006
head apoptotic process increased occurrence, abnormal WT + MO1-psenen Fig. 7Fig. 8 from Campbell et al., 2006
head cell death increased occurrence, abnormal WT + MO1-psenen Fig. 3 from Campbell et al., 2006
melanocyte migration disrupted, abnormal WT + MO1-psenen Fig. 3 from Ralser et al., 2017
midbrain hindbrain boundary absent, abnormal WT + MO1-psenen Fig. 3Fig. 8 from Campbell et al., 2006
Notch signaling pathway decreased occurrence, abnormal WT + MO1-psenen Fig. 4 from Campbell et al., 2006
pigment cell decreased amount, abnormal WT + MO1-psenen Fig. 3 from Ralser et al., 2017
pigment cell morphology, abnormal WT + MO1-psenen Fig. 3 from Ralser et al., 2017
pigmentation process quality, abnormal WT + MO1-psenen Fig. 3 from Ralser et al., 2017
post-vent region decreased length, abnormal WT + MO1-psenen Fig. 3 from Campbell et al., 2006
presumptive neural retina molecular quality, abnormal WT + MO1-psenen Fig. 4 from Campbell et al., 2006
primary motor neuron irregular spatial pattern, abnormal WT + MO1-psenen Fig. 3 from Campbell et al., 2006
Rohon-Beard neuron decreased amount, abnormal WT + MO1-psenen Fig. 9 from Campbell et al., 2006
somite shape, abnormal WT + MO1-psenen Fig. 5 from Campbell et al., 2006
somite border amorphous, abnormal WT + MO1-psenen Fig. 5 from Campbell et al., 2006
somitogenesis disrupted, abnormal WT + MO1-psenen Fig. 5 from Campbell et al., 2006
spinal cord has fewer parts of type primary motor neuron, abnormal WT + MO1-psenen Fig. 3 from Campbell et al., 2006
spinal cord primary motor neuron decreased amount, abnormal WT + MO1-psenen Fig. 9 from Campbell et al., 2006
spinal cord primary motor neuron spatial pattern, abnormal WT + MO1-psenen Fig. 9 from Campbell et al., 2006
trunk apoptotic process increased occurrence, abnormal WT + MO1-psenen Fig. 7 from Campbell et al., 2006
whole organism decreased size, abnormal WT + MO1-psenen Fig. 3Fig. 8 from Campbell et al., 2006
Phenotype of all Fish created by or utilizing MO1-psenen
Phenotype Fish Conditions Figures
brain primary motor neuron spatial pattern, abnormal WT + MO1-psenen standard conditions Fig. 9 from Campbell et al., 2006
spinal cord primary motor neuron decreased amount, abnormal WT + MO1-psenen standard conditions Fig. 9 from Campbell et al., 2006
somite border amorphous, abnormal WT + MO1-psenen standard conditions Fig. 5 from Campbell et al., 2006
post-vent region decreased length, abnormal WT + MO1-psenen standard conditions Fig. 3 from Campbell et al., 2006
spinal cord has fewer parts of type primary motor neuron, abnormal WT + MO1-psenen standard conditions Fig. 3 from Campbell et al., 2006
pigment cell decreased amount, abnormal WT + MO1-psenen standard conditions Fig. 3 from Ralser et al., 2017
Rohon-Beard neuron decreased amount, abnormal WT + MO1-psenen standard conditions Fig. 9 from Campbell et al., 2006
pigment cell morphology, abnormal WT + MO1-psenen standard conditions Fig. 3 from Ralser et al., 2017
trunk apoptotic process increased occurrence, abnormal WT + MO1-psenen standard conditions Fig. 7 from Campbell et al., 2006
somite shape, abnormal WT + MO1-psenen standard conditions Fig. 5 from Campbell et al., 2006
brain lacks parts or has fewer parts of type ventricular system, abnormal WT + MO1-psenen standard conditions Fig. 9 from Campbell et al., 2006
head decreased size, abnormal WT + MO1-psenen standard conditions Fig. 3 from Campbell et al., 2006
somitogenesis disrupted, abnormal WT + MO1-psenen standard conditions Fig. 5 from Campbell et al., 2006
midbrain hindbrain boundary absent, abnormal WT + MO1-psenen standard conditions Fig. 3Fig. 8 from Campbell et al., 2006
caudal fin increased pigmentation, abnormal WT + MO1-psenen standard conditions Fig. 3 from Ralser et al., 2017
head apoptotic process increased occurrence, abnormal WT + MO1-psenen standard conditions Fig. 7Fig. 8 from Campbell et al., 2006
whole organism decreased size, abnormal WT + MO1-psenen standard conditions Fig. 3Fig. 8 from Campbell et al., 2006
spinal cord primary motor neuron spatial pattern, abnormal WT + MO1-psenen standard conditions Fig. 9 from Campbell et al., 2006
primary motor neuron irregular spatial pattern, abnormal WT + MO1-psenen standard conditions Fig. 3 from Campbell et al., 2006
presumptive neural retina molecular quality, abnormal WT + MO1-psenen standard conditions Fig. 4 from Campbell et al., 2006
pigmentation process quality, abnormal WT + MO1-psenen standard conditions Fig. 3 from Ralser et al., 2017
brain primary motor neuron decreased amount, abnormal WT + MO1-psenen standard conditions Fig. 9 from Campbell et al., 2006
melanocyte migration disrupted, abnormal WT + MO1-psenen standard conditions Fig. 3 from Ralser et al., 2017
head cell death increased occurrence, abnormal WT + MO1-psenen standard conditions Fig. 3 from Campbell et al., 2006
head decreased pigmentation, abnormal WT + MO1-psenen standard conditions Fig. 3 from Ralser et al., 2017
Notch signaling pathway decreased occurrence, abnormal WT + MO1-psenen standard conditions Fig. 4 from Campbell et al., 2006
brain primary motor neuron spatial pattern, abnormal WT + MO1-psenen + MO4-tp53 standard conditions Fig. 9 from Campbell et al., 2006
whole organism decreased size, abnormal WT + MO1-psenen + MO4-tp53 standard conditions Fig. 8 from Campbell et al., 2006
brain primary motor neuron decreased amount, abnormal WT + MO1-psenen + MO4-tp53 standard conditions Fig. 9 from Campbell et al., 2006
Citations