Phenotype
|
Fish
|
Conditions
|
Figures
|
pronephric distal late tubule slc12a3 expression decreased distribution, abnormal
|
TU + MO2-ptger4a
|
standard conditions
|
Fig. 4
from Poureetezadi et al., 2016
|
pronephric distal late tubule decreased size, abnormal
|
TU + MO2-ptger4a
|
standard conditions
|
Fig. 4
from Poureetezadi et al., 2016
|
pronephric distal early tubule slc12a1 expression increased distribution, abnormal
|
TU + MO2-ptger4a
|
standard conditions
|
Fig. 4
from Poureetezadi et al., 2016
|
pronephric distal early tubule increased size, abnormal
|
TU + MO2-ptger4a
|
standard conditions
|
Fig. 4
from Poureetezadi et al., 2016
|
exocrine pancreas prss1 expression amount, ameliorated
|
WT + MO1-ptger4a + MO2-ptger4a
|
chemical treatment: prostaglandin E2
|
Fig. 2
from Nissim et al., 2014
|
exocrine pancreas prss1 expression decreased distribution, abnormal
|
WT + MO1-ptger4a + MO2-ptger4a
|
control
|
Fig. 2
from Nissim et al., 2014
|
exocrine pancreas size, ameliorated
|
WT + MO1-ptger4a + MO2-ptger4a
|
chemical treatment: prostaglandin E2
|
Fig. 2
from Nissim et al., 2014
|
hematopoietic stem cell decreased amount, abnormal
|
WT + MO1-ptger4a + MO2-ptger4a
|
standard conditions
|
Fig. S1
from North et al., 2007
|
exocrine pancreas decreased size, abnormal
|
WT + MO1-ptger4a + MO2-ptger4a
|
control
|
Fig. 2
from Nissim et al., 2014
|
Kupffer's vesicle motile cilium decreased length, abnormal
|
WT + MO2-ptger4a
|
chemical treatment: forskolin
|
Fig. 5
from Jin et al., 2014
|
trunk curved ventral, abnormal
|
WT + MO2-ptger4a
|
standard conditions
|
Fig. 5
from Jin et al., 2014
|
brain ventricular system edematous, abnormal
|
WT + MO2-ptger4a
|
chemical treatment: prostaglandin E2
|
Fig. 5
from Jin et al., 2014
|
Kupffer's vesicle motile cilium decreased length, abnormal
|
WT + MO2-ptger4a
|
chemical treatment: prostaglandin E2
|
Fig. 5
from Jin et al., 2014
|
cilium assembly decreased process quality, abnormal
|
WT + MO2-ptger4a
|
chemical treatment: prostaglandin E2
|
Fig. 5
from Jin et al., 2014
|
Kupffer's vesicle has fewer parts of type Kupffer's vesicle motile cilium, abnormal
|
WT + MO2-ptger4a
|
chemical treatment: forskolin
|
Fig. 5
from Jin et al., 2014
|
somite elongated, abnormal
|
WT + MO2-ptger4a
|
standard conditions
|
Fig. 3
from Cha et al., 2006
|
whole organism anterior-posterior axis decreased length, abnormal
|
WT + MO2-ptger4a
|
standard conditions
|
Fig. 3
from Cha et al., 2006
|
Kupffer's vesicle has fewer parts of type Kupffer's vesicle motile cilium, abnormal
|
WT + MO2-ptger4a
|
chemical treatment: prostaglandin E2
|
Fig. 5
from Jin et al., 2014
|
epiboly involved in gastrulation with mouth forming second delayed, abnormal
|
WT + MO2-ptger4a
|
standard conditions
|
Fig. 3
from Cha et al., 2006
|
heart bilateral symmetry, abnormal
|
WT + MO2-ptger4a
|
chemical treatment: prostaglandin E2
|
Fig. 5
from Jin et al., 2014
|
heart looping process quality, abnormal
|
WT + MO2-ptger4a
|
standard conditions
|
Fig. 5
from Jin et al., 2014
|
cilium assembly decreased process quality, abnormal
|
WT + MO2-ptger4a
|
standard conditions
|
Fig. 5
from Jin et al., 2014
|
heart bilateral symmetry, abnormal
|
WT + MO2-ptger4a
|
standard conditions
|
Fig. 5
from Jin et al., 2014
|
head position, abnormal
|
WT + MO2-ptger4a
|
standard conditions
|
Fig. 3
from Cha et al., 2006
|
axis decreased length, abnormal
|
WT + MO2-ptger4a
|
standard conditions
|
Fig. 3
from Cha et al., 2006
|
Kupffer's vesicle motile cilium decreased length, abnormal
|
WT + MO2-ptger4a
|
standard conditions
|
Fig. 5
from Jin et al., 2014
|
post-vent region curved ventral, abnormal
|
WT + MO2-ptger4a
|
standard conditions
|
Fig. 5
from Jin et al., 2014
|
ventral wall of dorsal aorta morphology, abnormal
|
WT + MO2-ptger4a
|
standard conditions
|
Fig. 5
from Villablanca et al., 2007
|
heart looping process quality, abnormal
|
WT + MO2-ptger4a
|
chemical treatment: prostaglandin E2
|
Fig. 5
from Jin et al., 2014
|
hemopoiesis disrupted, abnormal
|
WT + MO2-ptger4a
|
standard conditions
|
Fig. 5
from Villablanca et al., 2007
|
convergent extension involved in gastrulation disrupted, abnormal
|
WT + MO2-ptger4a
|
standard conditions
|
Fig. 3
from Cha et al., 2006
|
brain ventricular system edematous, abnormal
|
WT + MO2-ptger4a
|
standard conditions
|
Fig. 5
from Jin et al., 2014
|
brain hydrocephalic, abnormal
|
WT + MO2-ptger4a
|
standard conditions
|
Fig. 5
from Jin et al., 2014
|
Kupffer's vesicle has fewer parts of type Kupffer's vesicle motile cilium, abnormal
|
WT + MO2-ptger4a
|
standard conditions
|
Fig. 5
from Jin et al., 2014
|
notochord increased width, abnormal
|
WT + MO2-ptger4a
|
standard conditions
|
Fig. 3
from Cha et al., 2006
|
convergent extension involved in gastrulation delayed, abnormal
|
WT + MO2-ptger4a
|
standard conditions
|
Fig. 3
from Cha et al., 2006
|
otic vesicle has extra parts of type otolith, abnormal
|
WT + MO2-ptger4a
|
standard conditions
|
Fig. 5
from Jin et al., 2014
|
notochord undulate, abnormal
|
WT + MO2-ptger4a
|
standard conditions
|
Fig. 3
from Cha et al., 2006
|
lateral crista has fewer parts of type lateral crista kinocilium, abnormal
|
WT + MO2-ptger4a
|
standard conditions
|
Fig. 5
from Jin et al., 2014
|
liver EGFP expression decreased distribution, abnormal
|
as3Tg + MO1-ptger4a + MO2-ptger4a
|
control
|
Fig. 1
from Nissim et al., 2014
|
liver EGFP expression amount, ameliorated
|
as3Tg + MO1-ptger4a + MO2-ptger4a
|
chemical treatment: prostaglandin E2
|
Fig. 1
from Nissim et al., 2014
|
liver decreased size, abnormal
|
as3Tg + MO1-ptger4a + MO2-ptger4a
|
control
|
Fig. 1
from Nissim et al., 2014
|
liver size, ameliorated
|
as3Tg + MO1-ptger4a + MO2-ptger4a
|
chemical treatment: prostaglandin E2
|
Fig. 1
from Nissim et al., 2014
|
exocrine pancreas decreased size, abnormal
|
zf578Tg + MO1-ptger4a + MO2-ptger4a
|
control
|
Fig. 2
from Nissim et al., 2014
|
exocrine pancreas size, ameliorated
|
zf578Tg + MO1-ptger4a + MO2-ptger4a
|
chemical treatment: prostaglandin E2
|
Fig. 2
from Nissim et al., 2014
|
exocrine pancreas decreased size, abnormal
|
WT + MO1-ptger2a + MO1-ptger4a + MO2-ptger2a + MO2-ptger4a
|
chemical treatment: prostaglandin E2
|
Fig. 2
from Nissim et al., 2014
|
exocrine pancreas decreased size, abnormal
|
WT + MO1-ptger2a + MO1-ptger4a + MO2-ptger2a + MO2-ptger4a
|
control
|
Fig. 2
from Nissim et al., 2014
|
exocrine pancreas prss1 expression decreased distribution, abnormal
|
WT + MO1-ptger2a + MO1-ptger4a + MO2-ptger2a + MO2-ptger4a
|
control
|
Fig. 2
from Nissim et al., 2014
|
exocrine pancreas prss1 expression amount, ameliorated
|
WT + MO1-ptger2a + MO1-ptger4a + MO2-ptger2a + MO2-ptger4a
|
chemical treatment: prostaglandin E2
|
Fig. 2
from Nissim et al., 2014
|
liver decreased size, abnormal
|
as3Tg + MO1-ptger2a + MO1-ptger4a + MO2-ptger2a + MO2-ptger4a
|
chemical treatment: prostaglandin E2
|
Fig. 1
from Nissim et al., 2014
|
liver decreased size, abnormal
|
as3Tg + MO1-ptger2a + MO1-ptger4a + MO2-ptger2a + MO2-ptger4a
|
control
|
Fig. 1
from Nissim et al., 2014
|
exocrine pancreas decreased size, abnormal
|
zf578Tg + MO1-ptger2a + MO1-ptger4a + MO2-ptger2a + MO2-ptger4a
|
chemical treatment: prostaglandin E2
|
Fig. 2
from Nissim et al., 2014
|
exocrine pancreas decreased size, abnormal
|
zf578Tg + MO1-ptger2a + MO1-ptger4a + MO2-ptger2a + MO2-ptger4a
|
control
|
Fig. 2
from Nissim et al., 2014
|