HUMAN DISEASE MODELED by WT + MO1-mfn2

Human Disease	Conditions	Citations
Charcot-Marie-Tooth disease type 2	standard conditions	Vettori et al., 2011

GENE EXPRESSION
Gene expression in WT + MO1-mfn2

PHENOTYPE
Phenotype in WT + MO1-mfn2

Phenotype	Conditions	Figures
axon extension disrupted, abnormal	standard conditions	Fig. 3 from Vettori et al., 2011
brain edematous, abnormal	standard conditions	Fig. 2 from Vettori et al., 2011
eye decreased size, abnormal	standard conditions	Fig. 2 from Vettori et al., 2011
head decreased size, abnormal	standard conditions	Fig. 2 from Vettori et al., 2011
head necrotic, abnormal	standard conditions	Fig. 2 from Vettori et al., 2011
mandibular arch skeleton morphology, abnormal	standard conditions	Fig. 2 from Vettori et al., 2011
motor neuron axon absent, abnormal	standard conditions	Fig. 3 from Vettori et al., 2011
motor neuron axon branchiness, abnormal	standard conditions	Fig. 3 from Vettori et al., 2011
motor neuron axon truncated, abnormal	standard conditions	Fig. 3 from Vettori et al., 2011
post-vent region increased curvature, abnormal	standard conditions	Fig. 2 from Vettori et al., 2011
sensory perception of touch disrupted, abnormal	standard conditions	Fig. 2 from Vettori et al., 2011
skeletal muscle cell myofibril decreased thickness, abnormal	standard conditions	Fig. 5 from Vettori et al., 2011
skeletal muscle cell myofibril disorganized, abnormal	standard conditions	Fig. 5 from Vettori et al., 2011
somite decreased thickness, abnormal	standard conditions	Fig. 5 from Vettori et al., 2011
somite irregular spatial pattern, abnormal	standard conditions	Fig. 2 from Vettori et al., 2011
somite U-shaped, abnormal	standard conditions	Fig. 5 from Vettori et al., 2011
trunk edematous, abnormal	standard conditions	Fig. 2 from Vettori et al., 2011
whole organism decreased length, abnormal	standard conditions	Fig. 2 from Vettori et al., 2011
yolk distended, abnormal	standard conditions	Fig. 2 from Vettori et al., 2011