Phenotype
|
Conditions
|
Figures
|
adrenal gland development process quality, abnormal
|
standard conditions
|
Fig. 7
from Chou et al., 2014
|
anterior cardinal vein unlumenized, abnormal
|
standard conditions
|
Fig. 1
from Herbert et al., 2009
|
artery morphogenesis delayed, abnormal
|
standard conditions
|
Fig. 4
from Wang et al., 2011
|
basibranchial condensed, abnormal
|
standard conditions
|
Fig. 4
from Anderson et al., 2008
|
blood accumulation trunk, abnormal
|
standard conditions
|
Fig. 2
from Wang et al., 2011
|
blood circulation arrested, abnormal
|
standard conditions
|
Fig. 1
from Donat et al., 2018
Fig. 6
from Bussmann et al., 2011
Fig. 1
from Herbert et al., 2009
|
brain vasculature hemorrhagic, abnormal
|
standard conditions
|
Fig. S1
from Mugoni et al., 2013
|
cardiac muscle cell decreased size, normal
|
standard conditions
|
Fig. 7
from Yang et al., 2012
|
cardiac muscle cell electric potential, abnormal
|
standard conditions
|
Fig. 4
from Becker et al., 2011
|
cardiac muscle cell sarcomere disorganized, abnormal
|
standard conditions
|
Fig. 7
from Yang et al., 2012
|
caudal vein plexus klf2a expression decreased distribution, abnormal
|
standard conditions
|
Fig. 4
from Xie et al., 2018
|
caudal vein plexus kdrl expression decreased distribution, abnormal
|
control
|
Fig. 5
from Xie et al., 2018
|
central artery physical object quality, abnormal
|
standard conditions
|
Fig. 6
from Bussmann et al., 2011
|
ceratobranchial cartilage orientation ceratobranchial cartilage, abnormal
|
standard conditions
|
Fig. 4
from Anderson et al., 2008
|
ceratohyal cartilage orientation ceratohyal cartilage, abnormal
|
standard conditions
|
Fig. 4
from Anderson et al., 2008
|
chromaffin cell mislocalised, abnormal
|
standard conditions
|
Fig. 7
from Chou et al., 2014
|
common cardinal vein blood vessel endothelial cell decreased amount, abnormal
|
standard conditions
|
Fig. 8
from Helker et al., 2013
|
dorsal aorta dilated, abnormal
|
standard conditions
|
Fig. 4 ,
Fig. 7
from Ichimura et al., 2012
|
dorsal aorta lumenized, normal
|
standard conditions
|
Fig. 4
from Wang et al., 2011
Fig. 4
from Jin et al., 2007
|
dorsal aorta unlumenized, abnormal
|
standard conditions
|
Fig. 4
from Wang et al., 2011
|
endocardium morphology, normal
|
standard conditions
|
Fig. 6
from Serluca, 2008
|
endothelial cell amount, normal
|
standard conditions
|
Fig. 4
from Jin et al., 2007
|
heart heg1 expression decreased amount, abnormal
|
standard conditions
|
Fig. 1
from Fontana et al., 2020
|
heart flt1 expression decreased amount, abnormal
|
standard conditions
|
Fig. 1
from Fontana et al., 2020
|
heart notch1b expression decreased amount, abnormal
|
standard conditions
|
Fig. 1
from Fontana et al., 2020
|
heart flt4 expression increased amount, abnormal
|
standard conditions
|
Fig. 1
from Fontana et al., 2020
|
heart morphology, abnormal
|
standard conditions
|
Fig. S1
from Mugoni et al., 2013
|
hematopoietic multipotent progenitor cell decreased amount, abnormal
|
standard conditions
|
Fig. 7
from Wang et al., 2011
|
hematopoietic multipotent progenitor cell normal amount, normal
|
chemical treatment: S-nitroso-N-acetyl-D-penicillamine
|
Fig. 7
from Wang et al., 2011
|
interrenal gland animal organ morphogenesis process quality, abnormal
|
standard conditions
|
Fig. 7
from Chou et al., 2014
|
nitric oxide biosynthetic process disrupted, abnormal
|
standard conditions
|
Fig. 7
from Wang et al., 2011
|
nucleate erythrocyte development disrupted, abnormal
|
standard conditions
|
Fig. 8
from Helker et al., 2013
|
palatoquadrate arch decreased length, abnormal
|
standard conditions
|
Fig. 4
from Anderson et al., 2008
|
pericardium edematous, abnormal
|
standard conditions
|
Fig. 6
from Serluca, 2008
|
posterior cardinal vein lumenized, normal
|
standard conditions
|
Fig. 4
from Jin et al., 2007
|
posterior cardinal vein unlumenized, abnormal
|
standard conditions
|
Fig. 1
from Herbert et al., 2009
|
primitive erythrocyte differentiation process quality, normal
|
standard conditions
|
Fig. 1
from Wang et al., 2011
|
pronephric glomerulus disorganized, abnormal
|
standard conditions
|
Fig. 6 ,
Fig. 7
from Ichimura et al., 2012
|
pronephric glomerulus hypoplastic, abnormal
|
standard conditions
|
Fig. 4 ,
Fig. 7
from Ichimura et al., 2012
|
pronephric glomerulus lacks all parts of type pronephric glomerular capillary, abnormal
|
standard conditions
|
Fig. 4
from Ichimura et al., 2012
|
pronephric glomerulus morphology, abnormal
|
standard conditions
|
Fig. 4
from Ichimura et al., 2012
|
pronephric glomerulus unfused from pronephric glomerulus, abnormal
|
standard conditions
|
Fig. 4 ,
Fig. 7
from Ichimura et al., 2012
|
pronephric podocyte nphs1 expression spatial pattern, abnormal
|
standard conditions
|
Fig. 7
from Ichimura et al., 2012
|
pronephric podocyte podocyte foot present, normal
|
standard conditions
|
Fig. 6
from Ichimura et al., 2012
|
pronephric podocyte slit diaphragm present, normal
|
standard conditions
|
Fig. 6
from Ichimura et al., 2012
|
pronephros development process quality, abnormal
|
standard conditions
|
Fig. 4
from Ichimura et al., 2012
|
regulation of cardiac muscle contraction by calcium ion signaling disrupted, abnormal
|
standard conditions
|
Fig. 4
from Becker et al., 2011
|
sprouting angiogenesis process quality, abnormal
|
standard conditions
|
Fig. 6
from Bussmann et al., 2011
|
thymus decreased size, abnormal
|
standard conditions
|
Fig. 2
from Wang et al., 2011
|
thymus hematopoietic multipotent progenitor cell decreased amount, abnormal
|
standard conditions
|
Fig. 2
from Wang et al., 2011
|
trunk hematopoietic multipotent progenitor cell decreased amount, abnormal
|
standard conditions
|
Fig. 2,
Fig. 5
from Wang et al., 2011
|
trunk vasculature klf2a expression decreased amount, abnormal
|
standard conditions
|
Fig. S1
from Novodvorsky et al., 2015
|
venous blood vessel morphogenesis disrupted, abnormal
|
standard conditions
|
Fig. 1
from Herbert et al., 2009
|
ventral wall of dorsal aorta hematopoietic stem cell runx1 expression decreased amount, abnormal
|
standard conditions
|
Fig. S5
from Novodvorsky et al., 2015
|
ventral wall of dorsal aorta hematopoietic stem cell myb expression decreased amount, abnormal
|
standard conditions
|
Fig. S5
from Novodvorsky et al., 2015
|
ventricular myocardium decreased volume, normal
|
standard conditions
|
Fig. 7
from Yang et al., 2012
|
whole organism foxc1b expression decreased amount, abnormal
|
control
|
Fig. 6
from Chen et al., 2017
|
whole organism heg1 expression decreased amount, abnormal
|
standard conditions
|
Fig. 1
from Donat et al., 2018
|
whole organism foxc1a expression decreased amount, abnormal
|
control
|
Fig. 6
from Chen et al., 2017
|